Contralateral efferent inhibition amplifies interaural level differences

Evidence

In the MOC strategy, by contrast, c varied dynamically in time within the range [c max , c min ], where c max and c min produce the most and least amount of compression, respectively. For the natural MOCR, the more intense the contralateral stimulus, the greater the amount of efferent inhibition and the more linear the cochlear mechanical response (Hood et al. 1996 ). On the other hand, it seems reasonable that the amount of MOCR inhibition depends on the magnitude of the output from the cochlea rather than on the level of the acoustic stimulus. Inspired by this, we assumed that the instantaneous value of c for any given frequency channel in an MOC processor was inversely related to the output energy, E , from the corresponding channel in the contralateral MOC processor. In other words, the greater the output energy from a given frequency channel in the left-ear processor, the less the compression in the corresponding frequency channel of the right-ear processor, and vice versa (contralateral ‘on-frequency’ inhibition). — Dijk, Pim van, Physiology, Psychoacoustics and…, 1954 (#expands, via linked claim)

Categorization

• XREF: connects to: Interaural level differences of 1-2 dB produce just-audible lateral displacement — Professor Dr.-Ing. Hugo Fastl, Professor Dr.-Ing. Eberhard, Psychoacoustics- Facts and Models, 1714; Contralateral efferent inhibition of compression amplifies inter-aural level differences; Stronger contralateral sound causes greater efferent inhibition and more linear cochlear response — Dijk, Pim van, Physiology, Psychoacoustics and…, 1954 (+4 more)

Related

• Contralateral efferent inhibition of compression amplifies inter-aural level differences (similarity 0.94)
• Stronger contralateral sound causes greater efferent inhibition and more linear cochlear response (similarity 0.94)
• Contralateral dominance weakens at cortical stages due to commissural connections (similarity 0.93)
• Contralateral efferent inhibition is proportional to output energy, magnifying inter-aural differences. (similarity 0.92)
• The contralateral efferent reflex inhibits cochlear compression at matched frequencies (similarity 0.92)
• Contralateral efferent inhibition is nonlinear and output-dependent, amplifying asymmetries (similarity 0.91)
• The medial olivocochlear reflex inhibits cochlear compression contralaterally in a graded, output-dependent manner (similarity 0.88)
• Output-based cross-ear inhibition amplifies existing inter-aural asymmetries (similarity 0.88)
• Contralateral efferent-mediated processing can spatially segregate concurrent speech sources by ear (similarity 0.86)
• The contralateral efferent reflex strengthens gradually over tens of milliseconds (similarity 0.85)
• Interaural level differences of 1-2 dB produce just-audible lateral displacement (similarity 0.84)
• The real MOCR is stronger apically and inhibits a half-octave below the elicitor (similarity 0.84)
• Medial olivocochlear inhibition is stronger apically and shifted half an octave (similarity 0.84)
• Contralateral stimuli explain auditory brain activity better than ipsilateral ones (similarity 0.84)
• The stronger side suppresses the weaker side more, sharpening the lateral asymmetry. (similarity 0.84)

Broader Connections

• Contralateral efferent inhibition of compression amplifies inter-aural level differences #corroborates (0.98)
• Output-dependent inhibition creates amplificatory feedback that sharpens binaural asymmetries #expands (0.80)
• Stronger contralateral sound causes greater efferent inhibition and more linear cochlear response #expands (0.75) — Dijk, Pim van, Physiology, Psychoacoustics and…, 1954
• Contralateral efferent-mediated processing can spatially segregate concurrent speech sources by ear #expands (0.72)