The oscillating Bounce cannot be explained by ordinary acoustic overexposure damage

Evidence

Temporary or permanent damage to OHCs represent an unlikely cause, as recovery from acoustic overexposure with sounds in the sensitive range of hearing is typically monotonic, does not oscillate and no hypersensitivity can be seen. It has been shown, however, that intense sound stimulation of the cochlea in the isolated temporal bone preparation increases intracellular Ca2+ level of hair cells and supporting cells (Fridberger et al. 1998 ; Jacob et al. 2013 ). Acoustic injury consists of a plethora of structural and metabolic changes to the cochlea, with structural damage possibly masking more subtle (and possibly oscillating) metabolic changes of cochlear sensitivity caused by the rise of intracellular Ca2+ levels. — Dijk, Pim van, Physiology, Psychoacoustics and…, 3703

Categorization

• XREF: Distinguishes Bounce from classic noise-induced temporary threshold shift, which recovers monotonically.